Introduction
Unlike other patients, burn patients have a compromised natural skin barrier, leading to increased capillary permeability. This makes it easier for pathogenic microorganisms to penetrate through the broken skin or damaged intestinal mucosa, invading the organism. Additionally, burn wounds are accompanied by a large number of necrotic tissues and inflammatory exudate, which activate pro-inflammatory and anti-inflammatory signaling pathways. This dysregulation of the innate and adaptive immune response ultimately results in a high incidence of infections in burn patients.1–3 The incidence of infection in burn patients remains high, with potential consequences including delayed healing, scar tissue proliferation, bacteremia, sepsis, and multiple organ dysfunction syndrome once the pathogen invades the…