Introduction
In China, three prevalent types of neuronal surface antibody-associated autoimmune encephalitis (NSAE) include NMDA receptor encephalitis (NMDAR-E), leucine-rich glioma inactivated protein-1 encephalitis (LGI1-E), and gamma-aminobutyric acid-B receptor encephalitis (GABABR-E). Patients suffering from these NSAE subtypes display various clinical symptoms, such as seizures, psychiatric and behavioral issues, speech difficulties, autonomic nervous system dysfunction, cognitive impairments and involuntary movements.1,2 However, the precise mechanisms behind these NSAE subtypes are not fully understood. Research indicates that immune responses involving B cells may significantly contribute to the pathogenesis of these conditions.3,4 The detection of immunoglobulin G (IgG) oligoclonal bands (OCBs) is an immune…