Introduction
Anterior ischemic optic neuropathy without arteritis (NAION) represents a leading cause of optic nerve edema and dysfunction among individuals aged 50 and above.1–4 While the complete pathophysiological mechanism remains to be fully understood, current evidence suggests that reduced blood flow through the short posterior ciliary arteries to the optic nerve plays a central role in disease development.1,5 Recently, the focus on correlation between morphological indicators and functional condition has been increased. Ganglion cell–inner plexiform layer (GCIPL) analysis was reported as a useful biomarker for ganglion cell damage, which significantly correlated with best-corrected visual acuity (BCVA), visual field indices (mean deviation (MD) and visual field index (VFI)) in NAION patients.6,7 Nerve…